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Discussion

Blood pressure supplies blood to all parts of the body as it is pumped through the heart. The systolic and desolate pressure helps to facilitate this movement resulting from the ventricular relaxation and contraction. However, the primary reason hypertension sometimes may characterize the functioning of the heart. Primary hypertension can be detrimental because it results from an overactive SNS, overactive RAAS, and hormonal changes among obese individuals. These occur when there is increased diastolic pressure.

An individual with an overreactive SNS experienced a high possibility of developing paroxysmal sympathetic hyperreactivity. This is a condition associated with an increased risk of hypertension, especially because of our increasing activity with the sympathetic nervous system. This condition increases respiration rates and hatcheries that arise as primary symptoms of high blood pressure. The management of this condition requires incorporating diverse missions to increase the level of physical activities and healthy dietary practices (Sexana et al., 2018).

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An overactive renin-angiotensin-aldosterone system (RAAS) is also a major factor in high blood pressure. RAAS helps with the regulation of cardiovascular and renal functions. However, with primary high blood pressure, an increase in arterial pressure arises when the body fails to regulate extracellular fluid. When managing this condition, focused on incorporating both pharmacological and nonpharmacological measures focused on improving renal function.

For obesity-related hormonal changes, they affect the body’s general functioning because of the accumulation of fat in specific regions, which increases the risk of primary hypertension. This one of the changes is often defined by reducing parasympathetic tone and increased sympathetic activity. The negative effects are associated with numerous changes in outdoor genomic activity, contributing to a decline in heart rate variability. The best techniques of managing this condition will entail incorporating approaches aimed at reducing baroreflex sensitivity and individuals (Hall et al., 2015).

References

Hall, J. E., do Carmo, J. M., da Silva, A. A., Wang, Z., & Hall, M. E. (2015). Obesity-induced hypertension: interaction of neurohumoral and renal mechanisms. Circulation Research, 116(6), 991-1006.

Saxena, T., Ali, A. O., & Saxena, M. (2018). Pathophysiology of essential hypertension: an update. Expert Review of Cardiovascular Therapy, 16(12), 879-887.

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